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ey0016.8-5 | Important for Clinical Practice | ESPEYB16

8.5. Congenital adrenal hyperplasia due to steroid 21-hydroxylase deficiency: an endocrine society clinical practice guideline

PW Speiser , W Arlt , RJ Auchus , LS Baskin , GS Conway , DP Merke , HFL Meyer-Bahlburg , WL Miller , MH Murad , SE Oberfield , PC White

To read the full abstract: J Clin Endocrinol Metab. 2018; 103(11): 40434088.Since the publication of the 2010 Endocrine Society clinical practice guideline for Congenital Adrenal Hyperplasia (CAH) due to 21-hydroxylase deficiency, there have been several advances in the diagnosis and management of the condition. Neonatal diagnosis methods have been refined to use gestational age in...

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ey0015.5-9 | Life-long skeletal consequences of delayed puberty | ESPEYB15

5.9 Genetically Determined Later Puberty Impacts Lowered Bone Mineral Density in Childhood and Adulthood

DL Cousminer , JA Mitchell , A Chesi , SM Roy , HJ Kalkwarf , JM Lappe , V Gilsanz , SE Oberfield , JA Shepherd , A Kelly , SE McCormack , BF Voight , BS Zemel , SF Grant

To read the full abstract: J Bone Miner Res 2018;33:430-436Bone mass increases dramatically during puberty. The process of sexual maturation is therefore likely to impact lifelong bone health. Bone mineral density (BMD) tracks throughout life and later age at menarche is associated with increased osteoporosis risk in women, possibly because of combined effects of later menarche and lower peak bone ma...

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ESPE Yearbook of Paediatric Endocrinology

8.5. Congenital adrenal hyperplasia due to steroid 21-hydroxylase deficiency: an endocrine society clinical practice guideline

5.9 Genetically Determined Later Puberty Impacts Lowered Bone Mineral Density in Childhood and Adulthood

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